Oxidative Stress-Induced Unscheduled CDK1–Cyclin B1 Activity Impairs ER–Mitochondria-Mediated Bioenergetic Metabolism

Targeting the activities of endoplasmic reticulum (ER)–mitochondrial-dependent metabolic reprogramming is considered one of the most promising strategies for cancer treatment. Here, we present biochemical subcellular fractionation, coimmunoprecipitation, gene manipulation, and pharmacologic evidence...

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Detalles Bibliográficos
Autores principales: Chang, Jan-Gowth, Tien, Ni, Chang, Yi-Chih, Lin, Meng-Liang, Chen, Shih-Shun
Formato: Online Artículo Texto
Lenguaje:English
Publicado: MDPI 2021
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8224302/
https://www.ncbi.nlm.nih.gov/pubmed/34064109
http://dx.doi.org/10.3390/cells10061280

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8224302/
https://www.ncbi.nlm.nih.gov/pubmed/34064109
http://dx.doi.org/10.3390/cells10061280

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