Inhibition of Glycine Re-Uptake: A Potential Approach for Treating Pain by Augmenting Glycine-Mediated Spinal Neurotransmission and Blunting Central Nociceptive Signaling

Among the myriad of cellular and molecular processes identified as contributing to pathological pain, disinhibition of spinal cord nociceptive signaling to higher cortical centers plays a critical role. Importantly, evidence suggests that impaired glycinergic neurotransmission develops in the dorsal...

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Detalles Bibliográficos
Autor principal: Cioffi, Christopher L.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: MDPI 2021
Materias:
Review
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8230656/
https://www.ncbi.nlm.nih.gov/pubmed/34200954
http://dx.doi.org/10.3390/biom11060864

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8230656/
https://www.ncbi.nlm.nih.gov/pubmed/34200954
http://dx.doi.org/10.3390/biom11060864

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