MUC16 Is Overexpressed in Idiopathic Pulmonary Fibrosis and Induces Fibrotic Responses Mediated by Transforming Growth Factor-β1 Canonical Pathway

Several transmembrane mucins have demonstrated that they contribute intracellularly to induce fibrotic processes. The extracellular domain of MUC16 is considered as a biomarker for disease progression and death in IPF patients. However, there is no evidence regarding the signalling capabilities of M...

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Detalles Bibliográficos
Autores principales: Ballester, Beatriz, Milara, Javier, Montero, Paula, Cortijo, Julio
Formato: Online Artículo Texto
Lenguaje:English
Publicado: MDPI 2021
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8235375/
https://www.ncbi.nlm.nih.gov/pubmed/34204432
http://dx.doi.org/10.3390/ijms22126502

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8235375/
https://www.ncbi.nlm.nih.gov/pubmed/34204432
http://dx.doi.org/10.3390/ijms22126502

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