Difluoromethylornithine attenuates isoproterenol-induced cardiac hypertrophy by regulating apoptosis, autophagy and the mitochondria-associated membranes pathway

Myocardial hypertrophy is an independent risk factor of cardiovascular diseases and is closely associated with the incidence of heart failure. In the present study, we hypothesized that difluoromethylornithine (DFMO) could attenuate cardiac hypertrophy through mitochondria-associated membranes (MAM)...

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Detalles Bibliográficos
Autores principales: Zhao, Yu, Jia, Wei-Wei, Ren, San, Xiao, Wei, Li, Guang-Wei, Jin, Li, Lin, Yan
Formato: Online Artículo Texto
Lenguaje:English
Publicado: D.A. Spandidos 2021
Materias:
Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8237397/
https://www.ncbi.nlm.nih.gov/pubmed/34194548
http://dx.doi.org/10.3892/etm.2021.10302

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8237397/
https://www.ncbi.nlm.nih.gov/pubmed/34194548
http://dx.doi.org/10.3892/etm.2021.10302

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