Endostatin attenuates heart failure via inhibiting reactive oxygen species in myocardial infarction rats

The purpose of the present study was to evaluate whether endostatin overexpression could improve cardiac function, hemodynamics, and fibrosis in heart failure (HF) via inhibiting reactive oxygen species (ROS). The HF models were established by inducing ischemia myocardial infarction (MI) through lig...

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Detalles Bibliográficos
Autores principales: Xu, Xuguang, Jiang, Tingbo, Li, Yong, Kong, Liusha
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Portland Press Ltd. 2021
Materias:
Cardiovascular System & Vascular Biology
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8243342/
https://www.ncbi.nlm.nih.gov/pubmed/32686821
http://dx.doi.org/10.1042/BSR20200787

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8243342/
https://www.ncbi.nlm.nih.gov/pubmed/32686821
http://dx.doi.org/10.1042/BSR20200787

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