Effect of the KCa3.1 blocker, senicapoc, on cerebral edema and cardiovascular function after cardiac arrest — A randomized experimental rat study

AIM: Formation of cerebral edema and cardiovascular dysfunction may worsen brain injury following cardiac arrest. We hypothesized that administration of the intermediate calcium-activated potassium (KCa3.1) channel blocker, senicapoc, would reduce cerebral edema and augment mean arterial pressure in...

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Detalles Bibliográficos
Autores principales: Hansen, Frederik Boe, Secher, Niels, Mattson, Thomas, Løfgren, Bo, Simonsen, Ulf, Granfeldt, Asger
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Elsevier 2021
Materias:
Experimental Paper
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8244250/
https://www.ncbi.nlm.nih.gov/pubmed/34223371
http://dx.doi.org/10.1016/j.resplu.2021.100111

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8244250/
https://www.ncbi.nlm.nih.gov/pubmed/34223371
http://dx.doi.org/10.1016/j.resplu.2021.100111

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