Targeting mitochondrial reactive oxygen species-mediated oxidative stress attenuates nicotine-induced cardiac remodeling and dysfunction

Long-term nicotine intake is associated with an increased risk of myocardial damage and dysfunction. However, it remains unclear whether targeting mitochondrial reactive oxygen species (ROS) prevents nicotine-induced cardiac remodeling and dysfunction. This study investigated the effects of mitoTEMP...

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Detalles Bibliográficos
Autores principales: Ramalingam, Anand, Budin, Siti Balkis, Mohd Fauzi, Norsyahida, Ritchie, Rebecca H., Zainalabidin, Satirah
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Nature Publishing Group UK 2021
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8257608/
https://www.ncbi.nlm.nih.gov/pubmed/34226619
http://dx.doi.org/10.1038/s41598-021-93234-4

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8257608/
https://www.ncbi.nlm.nih.gov/pubmed/34226619
http://dx.doi.org/10.1038/s41598-021-93234-4

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