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Targeting mitochondrial reactive oxygen species-mediated oxidative stress attenuates nicotine-induced cardiac remodeling and dysfunction
Long-term nicotine intake is associated with an increased risk of myocardial damage and dysfunction. However, it remains unclear whether targeting mitochondrial reactive oxygen species (ROS) prevents nicotine-induced cardiac remodeling and dysfunction. This study investigated the effects of mitoTEMP...
Autores principales: | Ramalingam, Anand, Budin, Siti Balkis, Mohd Fauzi, Norsyahida, Ritchie, Rebecca H., Zainalabidin, Satirah |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Nature Publishing Group UK
2021
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8257608/ https://www.ncbi.nlm.nih.gov/pubmed/34226619 http://dx.doi.org/10.1038/s41598-021-93234-4 |
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