ERα36–GPER1 Collaboration Inhibits TLR4/NFκB-Induced Pro-Inflammatory Activity in Breast Cancer Cells

Inflammation is important for the initiation and progression of breast cancer. We have previously reported that in monocytes, estrogen regulates TLR4/NFκB-mediated inflammation via the interaction of the Erα isoform ERα36 with GPER1. We therefore investigated whether a similar mechanism is present i...

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Detalles Bibliográficos
Autores principales: Notas, George, Panagiotopoulos, Athanasios, Vamvoukaki, Rodanthi, Kalyvianaki, Konstantina, Kiagiadaki, Foteini, Deli, Alexandra, Kampa, Marilena, Castanas, Elias
Formato: Online Artículo Texto
Lenguaje:English
Publicado: MDPI 2021
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8303269/
https://www.ncbi.nlm.nih.gov/pubmed/34299224
http://dx.doi.org/10.3390/ijms22147603

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8303269/
https://www.ncbi.nlm.nih.gov/pubmed/34299224
http://dx.doi.org/10.3390/ijms22147603

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