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ERα36–GPER1 Collaboration Inhibits TLR4/NFκB-Induced Pro-Inflammatory Activity in Breast Cancer Cells
Inflammation is important for the initiation and progression of breast cancer. We have previously reported that in monocytes, estrogen regulates TLR4/NFκB-mediated inflammation via the interaction of the Erα isoform ERα36 with GPER1. We therefore investigated whether a similar mechanism is present i...
Autores principales: | Notas, George, Panagiotopoulos, Athanasios, Vamvoukaki, Rodanthi, Kalyvianaki, Konstantina, Kiagiadaki, Foteini, Deli, Alexandra, Kampa, Marilena, Castanas, Elias |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
MDPI
2021
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8303269/ https://www.ncbi.nlm.nih.gov/pubmed/34299224 http://dx.doi.org/10.3390/ijms22147603 |
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