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K(v)1.1 channels mediate network excitability and feed-forward inhibition in local amygdala circuits
K(v)1.1 containing potassium channels play crucial roles towards dampening neuronal excitability. Mice lacking K(v)1.1 subunits (Kcna1(−/−)) display recurrent spontaneous seizures and often exhibit sudden unexpected death. Seizures in Kcna1(−/−) mice resemble those in well-characterized models of te...
| Autores principales: | , , , |
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| Formato: | Online Artículo Texto |
| Lenguaje: | English |
| Publicado: |
Nature Publishing Group UK
2021
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| Materias: | |
| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8313690/ https://www.ncbi.nlm.nih.gov/pubmed/34312446 http://dx.doi.org/10.1038/s41598-021-94633-3 |