AQP3‐mediated H(2)O(2) uptake inhibits LUAD autophagy by inactivating PTEN

It is widely accepted that redox reprogramming participates in malignant transformation of lung adenocarcinoma (LUAD). However, the source of excessive reactive oxygen species (ROS) and the downstream signaling regulatory mechanism are complicated and unintelligible. In the current study, we newly i...

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Detalles Bibliográficos
Autores principales: Wang, Yawei, Chen, Di, Liu, Yu, Zhang, Yong, Duan, Chao, Otkur, Wuxiyar, Chen, Huan, Liu, Xiaolong, Xia, Tian, Qi, Huan, Piao, Hai‐long, Liu, Hong‐Xu
Formato: Online Artículo Texto
Lenguaje:English
Publicado: John Wiley and Sons Inc. 2021
Materias:
Original Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8353907/
https://www.ncbi.nlm.nih.gov/pubmed/34091997
http://dx.doi.org/10.1111/cas.15008

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8353907/
https://www.ncbi.nlm.nih.gov/pubmed/34091997
http://dx.doi.org/10.1111/cas.15008

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