MKP-3 suppresses LPS-induced inflammatory responses in HUVECs via inhibition of p38 MAPK/NF-κB pathway

Endothelial cell dysfunction and inflammatory responses play critical roles in the development of atherosclerosis. Recent data on the processes underlying atherogenesis indicate the substantial role of endotoxins (lipopolysaccharides; LPS) of the intestinal microflora in the initiation and progressi...

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Detalles Bibliográficos
Autores principales: Unenkhuu, Banzragchgarav, Kim, Da Bin, Kim, Hong Seok
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Taylor & Francis 2021
Materias:
Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8366647/
https://www.ncbi.nlm.nih.gov/pubmed/34408812
http://dx.doi.org/10.1080/19768354.2021.1954551

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8366647/
https://www.ncbi.nlm.nih.gov/pubmed/34408812
http://dx.doi.org/10.1080/19768354.2021.1954551

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