Platelet P2Y (12) Receptor Deletion or Pharmacological Inhibition does not Protect Mice from Sepsis or Septic Shock

Introduction  Platelets are increasingly appreciated as key effectors during sepsis, raising the question of the usefulness of antiplatelet drugs to treat patients with sepsis. Objective  Evaluate the potential contribution of the platelet P2Y (12) receptor in the pathogenesis of polymicrobial-induc...

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Detalles Bibliográficos
Autores principales: Rabouel, Yannick, Magnenat, Stéphanie, Delabranche, Xavier, Gachet, Christian, Hechler, Beatrice
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Georg Thieme Verlag KG 2021
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8384481/
https://www.ncbi.nlm.nih.gov/pubmed/34447900
http://dx.doi.org/10.1055/s-0041-1733857

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8384481/
https://www.ncbi.nlm.nih.gov/pubmed/34447900
http://dx.doi.org/10.1055/s-0041-1733857

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