The histone chaperone Anp32e regulates memory formation, transcription, and dendritic morphology by regulating steady-state H2A.Z binding in neurons

Rapid removal of histone H2A.Z from neuronal chromatin is a key step in learning-induced gene expression and memory formation, but mechanisms underlying learning-induced H2A.Z removal are unclear. Anp32e was recently identified as an H2A.Z-specific histone chaperone that removes H2A.Z from nucleosom...

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Detalles Bibliográficos
Autores principales: Stefanelli, Gilda, Makowski, Claire E., Brimble, Mark A., Hall, Meaghan, Reda, Anas, Creighton, Samantha D., Leonetti, Amanda M., McLean, Timothy A.B., Zakaria, Jacqueline M., Baumbach, Jennet, Greer, Celeste B., Davidoff, Andrew M., Walters, Brandon J., Murphy, Patrick J., Zovkic, Iva B.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: 2021
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8422973/
https://www.ncbi.nlm.nih.gov/pubmed/34407406
http://dx.doi.org/10.1016/j.celrep.2021.109551

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8422973/
https://www.ncbi.nlm.nih.gov/pubmed/34407406
http://dx.doi.org/10.1016/j.celrep.2021.109551

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