Interleukin‐1β exacerbates disease and is a potential therapeutic target to reduce pulmonary inflammation during severe influenza A virus infection

Hyperinflammatory responses including the production of NLRP3‐dependent interleukin (IL)‐1β is a characteristic feature of severe and fatal influenza A virus (IAV) infections. The NLRP3 inflammasome has been shown to play a temporal role during severe IAV immune responses, with early protective and...

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Detalles Bibliográficos
Autores principales: Bawazeer, Abdulah OS, Rosli, Sarah, Harpur, Christopher M, Docherty, Callum AH, Mansell, Ashley, Tate, Michelle D
Formato: Online Artículo Texto
Lenguaje:English
Publicado: John Wiley and Sons Inc. 2021
Materias:
Original Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8453884/
https://www.ncbi.nlm.nih.gov/pubmed/33834544
http://dx.doi.org/10.1111/imcb.12459

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8453884/
https://www.ncbi.nlm.nih.gov/pubmed/33834544
http://dx.doi.org/10.1111/imcb.12459

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