KLF4 Promotes Diabetic Chronic Wound Healing by Suppressing Th17 Cell Differentiation in an MDSC-Dependent Manner

OBJECTIVES: Diabetic wound inflammation deficiencies lead to ulcer development and eventual amputation and disability. Our previous research demonstrates that myeloid-derived suppressor cells (MDSCs) accumulate during inflammation and promote chronic wound healing via the regulation of Kruppel-like...

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Detalles Bibliográficos
Autores principales: Yang, Xiong, Mathis, Bryan J., Huang, Yu, Li, Wencheng, Shi, Ying
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Hindawi 2021
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8457977/
https://www.ncbi.nlm.nih.gov/pubmed/34568499
http://dx.doi.org/10.1155/2021/7945117

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8457977/
https://www.ncbi.nlm.nih.gov/pubmed/34568499
http://dx.doi.org/10.1155/2021/7945117

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