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KLF4 Promotes Diabetic Chronic Wound Healing by Suppressing Th17 Cell Differentiation in an MDSC-Dependent Manner
OBJECTIVES: Diabetic wound inflammation deficiencies lead to ulcer development and eventual amputation and disability. Our previous research demonstrates that myeloid-derived suppressor cells (MDSCs) accumulate during inflammation and promote chronic wound healing via the regulation of Kruppel-like...
Autores principales: | Yang, Xiong, Mathis, Bryan J., Huang, Yu, Li, Wencheng, Shi, Ying |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Hindawi
2021
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8457977/ https://www.ncbi.nlm.nih.gov/pubmed/34568499 http://dx.doi.org/10.1155/2021/7945117 |
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