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Targeting the Pentose Phosphate Pathway for SARS-CoV-2 Therapy
SARS-CoV-2 is causing the coronavirus disease 2019 (COVID-19) pandemic, for which effective pharmacological therapies are needed. SARS-CoV-2 induces a shift of the host cell metabolism towards glycolysis, and the glycolysis inhibitor 2-deoxy-d-glucose (2DG), which interferes with SARS-CoV-2 infectio...
Autores principales: | , , , , , , , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
MDPI
2021
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8540749/ https://www.ncbi.nlm.nih.gov/pubmed/34677415 http://dx.doi.org/10.3390/metabo11100699 |
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author | Bojkova, Denisa Costa, Rui Reus, Philipp Bechtel, Marco Jaboreck, Mark-Christian Olmer, Ruth Martin, Ulrich Ciesek, Sandra Michaelis, Martin Cinatl, Jindrich |
author_facet | Bojkova, Denisa Costa, Rui Reus, Philipp Bechtel, Marco Jaboreck, Mark-Christian Olmer, Ruth Martin, Ulrich Ciesek, Sandra Michaelis, Martin Cinatl, Jindrich |
author_sort | Bojkova, Denisa |
collection | PubMed |
description | SARS-CoV-2 is causing the coronavirus disease 2019 (COVID-19) pandemic, for which effective pharmacological therapies are needed. SARS-CoV-2 induces a shift of the host cell metabolism towards glycolysis, and the glycolysis inhibitor 2-deoxy-d-glucose (2DG), which interferes with SARS-CoV-2 infection, is under development for the treatment of COVID-19 patients. The glycolytic pathway generates intermediates that supply the non-oxidative branch of the pentose phosphate pathway (PPP). In this study, the analysis of proteomics data indicated increased transketolase (TKT) levels in SARS-CoV-2-infected cells, suggesting that a role is played by the non-oxidative PPP. In agreement, the TKT inhibitor benfooxythiamine (BOT) inhibited SARS-CoV-2 replication and increased the anti-SARS-CoV-2 activity of 2DG. In conclusion, SARS-CoV-2 infection is associated with changes in the regulation of the PPP. The TKT inhibitor BOT inhibited SARS-CoV-2 replication and increased the activity of the glycolysis inhibitor 2DG. Notably, metabolic drugs like BOT and 2DG may also interfere with COVID-19-associated immunopathology by modifying the metabolism of immune cells in addition to inhibiting SARS-CoV-2 replication. Hence, they may improve COVID-19 therapy outcomes by exerting antiviral and immunomodulatory effects. |
format | Online Article Text |
id | pubmed-8540749 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2021 |
publisher | MDPI |
record_format | MEDLINE/PubMed |
spelling | pubmed-85407492021-10-24 Targeting the Pentose Phosphate Pathway for SARS-CoV-2 Therapy Bojkova, Denisa Costa, Rui Reus, Philipp Bechtel, Marco Jaboreck, Mark-Christian Olmer, Ruth Martin, Ulrich Ciesek, Sandra Michaelis, Martin Cinatl, Jindrich Metabolites Article SARS-CoV-2 is causing the coronavirus disease 2019 (COVID-19) pandemic, for which effective pharmacological therapies are needed. SARS-CoV-2 induces a shift of the host cell metabolism towards glycolysis, and the glycolysis inhibitor 2-deoxy-d-glucose (2DG), which interferes with SARS-CoV-2 infection, is under development for the treatment of COVID-19 patients. The glycolytic pathway generates intermediates that supply the non-oxidative branch of the pentose phosphate pathway (PPP). In this study, the analysis of proteomics data indicated increased transketolase (TKT) levels in SARS-CoV-2-infected cells, suggesting that a role is played by the non-oxidative PPP. In agreement, the TKT inhibitor benfooxythiamine (BOT) inhibited SARS-CoV-2 replication and increased the anti-SARS-CoV-2 activity of 2DG. In conclusion, SARS-CoV-2 infection is associated with changes in the regulation of the PPP. The TKT inhibitor BOT inhibited SARS-CoV-2 replication and increased the activity of the glycolysis inhibitor 2DG. Notably, metabolic drugs like BOT and 2DG may also interfere with COVID-19-associated immunopathology by modifying the metabolism of immune cells in addition to inhibiting SARS-CoV-2 replication. Hence, they may improve COVID-19 therapy outcomes by exerting antiviral and immunomodulatory effects. MDPI 2021-10-13 /pmc/articles/PMC8540749/ /pubmed/34677415 http://dx.doi.org/10.3390/metabo11100699 Text en © 2021 by the authors. https://creativecommons.org/licenses/by/4.0/Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/). |
spellingShingle | Article Bojkova, Denisa Costa, Rui Reus, Philipp Bechtel, Marco Jaboreck, Mark-Christian Olmer, Ruth Martin, Ulrich Ciesek, Sandra Michaelis, Martin Cinatl, Jindrich Targeting the Pentose Phosphate Pathway for SARS-CoV-2 Therapy |
title | Targeting the Pentose Phosphate Pathway for SARS-CoV-2 Therapy |
title_full | Targeting the Pentose Phosphate Pathway for SARS-CoV-2 Therapy |
title_fullStr | Targeting the Pentose Phosphate Pathway for SARS-CoV-2 Therapy |
title_full_unstemmed | Targeting the Pentose Phosphate Pathway for SARS-CoV-2 Therapy |
title_short | Targeting the Pentose Phosphate Pathway for SARS-CoV-2 Therapy |
title_sort | targeting the pentose phosphate pathway for sars-cov-2 therapy |
topic | Article |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8540749/ https://www.ncbi.nlm.nih.gov/pubmed/34677415 http://dx.doi.org/10.3390/metabo11100699 |
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