GAP-43 ameliorates Podocyte injury by decreasing nuclear NFATc1 expression

Podocyte injury is sufficient to cause glomerulosclerosis and proteinuria, eventually leading to kidney failure. Previous studies found that podocytes and neurons had similar biological characteristics. Growth-associated protein-43 (GAP-43) is a growth cone protein in neurons, and a marker of axonal...

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Detalles Bibliográficos
Autores principales: Lian, Zhiwen, Ke, Guibao, Zhang, Hong, Dou, Caoshuai, Chen, Xueqin, Li, Bohou, Zhang, Fengxia, Wen, Shichun, Wu, Qiong, Xia, Yubin, Jiang, Nan, Li, Zhuo, Li, Sijia, Zhao, Xingchen, Ma, Jianchao, Lin, Ting, Wen, Feng, Xu, Lixia, Li, Zhilian, Liang, Huabang, Dong, Wei, Chen, Yuanhan, Li, Ruizhao, Ye, Zhiming, Wang, Wenjian, Liang, Xinling, Shi, Wei, Zhang, Li, Liu, Shuangxin
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Elsevier 2021
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8551842/
https://www.ncbi.nlm.nih.gov/pubmed/34746448
http://dx.doi.org/10.1016/j.bbrep.2021.101145

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8551842/
https://www.ncbi.nlm.nih.gov/pubmed/34746448
http://dx.doi.org/10.1016/j.bbrep.2021.101145

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