Toll-Like Receptor-4 Antagonist Enhances the Repair of Ultraviolet Radiation-Induced DNA Damage and Augments Anti-Tumor Immune Responses in Mice

SIMPLE SUMMARY: Ultraviolet B (UVB) radiation is largely responsible for the development of skin cancer. When UVB-induced DNA damage in cells is not repaired, it can lead to the initiation of non-melanoma skin cancers. Xeroderma pigmentosum (XP) disease is caused by a defect in the repair of damaged...

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Detalles Bibliográficos
Autores principales: Sherwani, Mohammad Asif, Abdelgawad, Ahmed, Chung, Minh, Ibrahim, Saad, Eraslan, Mualla, Elmets, Craig A., Yusuf, Nabiha
Formato: Online Artículo Texto
Lenguaje:English
Publicado: MDPI 2021
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8582386/
https://www.ncbi.nlm.nih.gov/pubmed/34771569
http://dx.doi.org/10.3390/cancers13215406

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8582386/
https://www.ncbi.nlm.nih.gov/pubmed/34771569
http://dx.doi.org/10.3390/cancers13215406

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