RIP3 Inhibition ameliorates chronic constriction injury-induced neuropathic pain by suppressing JNK signaling

Neuroinflammation is a major contributor to neuropathic pain. Receptor interacting serine/threonine kinase 3 (RIP3) senses cellular stress, promotes inflammatory responses and activates c-Jun N-terminal kinase (JNK) signaling. Here, we assessed the involvement of RIP3-induced JNK signaling in chroni...

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Detalles Bibliográficos
Autores principales: He, Na, Qu, Yu-Juan, Li, Dan-Yang, Yue, Shou-Wei
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Impact Journals 2021
Materias:
Research Paper
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8610111/
https://www.ncbi.nlm.nih.gov/pubmed/34772825
http://dx.doi.org/10.18632/aging.203691

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8610111/
https://www.ncbi.nlm.nih.gov/pubmed/34772825
http://dx.doi.org/10.18632/aging.203691

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