Anisomycin protects against sepsis by attenuating IκB kinase-dependent NF-κB activation and inflammatory gene expression

Anisomycin is known to inhibit eukaryotic protein synthesis and has been established as an antibiotic and anticancer drug. However, the molecular targets of anisomycin and its mechanism of action have not been explained in macrophages. Here, we demonstrated the anti-inflammatory effects of anisomyci...

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Detalles Bibliográficos
Autores principales: Park, Gyoung Lim, Park, Minkyung, Min, Jeong-Ki, Park, Young-Jun, Chung, Su Wol, Lee, Seon-Jin
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Korean Society for Biochemistry and Molecular Biology 2021
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8633526/
https://www.ncbi.nlm.nih.gov/pubmed/34353427
http://dx.doi.org/10.5483/BMBRep.2021.54.11.063

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8633526/
https://www.ncbi.nlm.nih.gov/pubmed/34353427
http://dx.doi.org/10.5483/BMBRep.2021.54.11.063

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