Intricate crosstalk between MYB and noncoding RNAs in cancer

MYB is often overexpressed in malignant tumors and plays a carcinogenic role in the initiation and development of cancer. Deletion of the MYB regulatory C-terminal domain may be a driving mutation leading to tumorigenesis, therefore, different tumor mechanisms produce similar MYB proteins. As MYB is...

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Detalles Bibliográficos
Autores principales: Hu, Dingyu, Shao, Wenjun, Liu, Li, Wang, Yanyan, Yuan, Shunling, Liu, Zhaoping, Liu, Jing, Zhang, Ji
Formato: Online Artículo Texto
Lenguaje:English
Publicado: BioMed Central 2021
Materias:
Review
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8650324/
https://www.ncbi.nlm.nih.gov/pubmed/34876130
http://dx.doi.org/10.1186/s12935-021-02362-4

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8650324/
https://www.ncbi.nlm.nih.gov/pubmed/34876130
http://dx.doi.org/10.1186/s12935-021-02362-4

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