Delayed induction of type I and III interferons mediates nasal epithelial cell permissiveness to SARS-CoV-2

The nasal epithelium is a plausible entry point for SARS-CoV-2, a site of pathogenesis and transmission, and may initiate the host response to SARS-CoV-2. Antiviral interferon (IFN) responses are critical to outcome of SARS-CoV-2. Yet little is known about the interaction between SARS-CoV-2 and inna...

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Detalles Bibliográficos
Autores principales: Hatton, Catherine F., Botting, Rachel A., Dueñas, Maria Emilia, Haq, Iram J., Verdon, Bernard, Thompson, Benjamin J., Spegarova, Jarmila Stremenova, Gothe, Florian, Stephenson, Emily, Gardner, Aaron I., Murphy, Sandra, Scott, Jonathan, Garnett, James P., Carrie, Sean, Powell, Jason, Khan, C. M. Anjam, Huang, Lei, Hussain, Rafiqul, Coxhead, Jonathan, Davey, Tracey, Simpson, A. John, Haniffa, Muzlifah, Hambleton, Sophie, Brodlie, Malcolm, Ward, Chris, Trost, Matthias, Reynolds, Gary, Duncan, Christopher J. A.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Nature Publishing Group UK 2021
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8651658/
https://www.ncbi.nlm.nih.gov/pubmed/34876592
http://dx.doi.org/10.1038/s41467-021-27318-0

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8651658/
https://www.ncbi.nlm.nih.gov/pubmed/34876592
http://dx.doi.org/10.1038/s41467-021-27318-0

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