Absence of PEXEL-Dependent Protein Export in Plasmodium Liver Stages Cannot Be Restored by Gain of the HSP101 Protein Translocon ATPase

Host cell remodeling is critical for successful Plasmodium replication inside erythrocytes and achieved by targeted export of parasite-encoded proteins. In contrast, during liver infection the malarial parasite appears to avoid protein export, perhaps to limit exposure of parasite antigens by infect...

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Detalles Bibliográficos
Autores principales: Kreutzfeld, Oriana, Grützke, Josephine, Ingmundson, Alyssa, Müller, Katja, Matuschewski, Kai
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Frontiers Media S.A. 2021
Materias:
Genetics
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8693896/
https://www.ncbi.nlm.nih.gov/pubmed/34956312
http://dx.doi.org/10.3389/fgene.2021.742153

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8693896/
https://www.ncbi.nlm.nih.gov/pubmed/34956312
http://dx.doi.org/10.3389/fgene.2021.742153

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