CaMKIIδ post-translational modifications increase affinity for calmodulin inside cardiac ventricular myocytes

Persistent over-activation of CaMKII (Calcium/Calmodulin-dependent protein Kinase II) in the heart is implicated in arrhythmias, heart failure, pathological remodeling, and other cardiovascular diseases. Several post-translational modifications (PTMs)—including autophosphorylation, oxidation, S-nitr...

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Detalles Bibliográficos
Autores principales: Simon, Mitchell, Ko, Christopher Y., Rebbeck, Robyn T., Baidar, Sonya, Cornea, Razvan L., Bers, Donald M.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: 2021
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8716136/
https://www.ncbi.nlm.nih.gov/pubmed/34371035
http://dx.doi.org/10.1016/j.yjmcc.2021.08.002

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8716136/
https://www.ncbi.nlm.nih.gov/pubmed/34371035
http://dx.doi.org/10.1016/j.yjmcc.2021.08.002

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