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A novel antagonist of TRPM2 and TRPV4 channels: Carvacrol
The overload cytosolic free Ca(2+) (cCa(2+)) influx-mediated excessive generation of oxidative stress in the pathophysiological conditions induces neuronal and cellular injury via the activation of cation channels. TRPM2 and TRPV4 channels are activated by oxidative stress, and their specific antago...
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| Formato: | Online Artículo Texto |
| Lenguaje: | English |
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Springer US
2022
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| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8732973/ https://www.ncbi.nlm.nih.gov/pubmed/34989943 http://dx.doi.org/10.1007/s11011-021-00887-1 |