Disrupted mitochondrial homeostasis coupled with mitotic arrest generates antineoplastic oxidative stress

Reactive oxygen species (ROS) serve as critical signals in various cellular processes. Excessive ROS cause cell death or senescence and mediates the therapeutic effect of many cancer drugs. Recent studies showed that ROS increasingly accumulate during G2/M arrest, the underlying mechanism, however,...

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Detalles Bibliográficos
Autores principales: Hao, Xiaohe, Bu, Wenqing, Lv, Guosheng, Xu, Limei, Hou, Dong, Wang, Jing, Liu, Xiaojie, Yang, Tingting, Zhang, Xiyu, Liu, Qiao, Gong, Yaoqin, Shao, Changshun
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Nature Publishing Group UK 2021
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8755538/
https://www.ncbi.nlm.nih.gov/pubmed/34773075
http://dx.doi.org/10.1038/s41388-021-02105-9

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8755538/
https://www.ncbi.nlm.nih.gov/pubmed/34773075
http://dx.doi.org/10.1038/s41388-021-02105-9

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