Sympathetic Stimulation Upregulates the Ca(2+) Channel Subunit, Ca(V)α2δ1, via the β1 and ERK 1/2 Pathway in Neonatal Ventricular Cardiomyocytes

Intracellular Ca(2+) overload secondary to chronic hemodynamic stimuli promotes the recruitment of Ca(2+)-dependent signaling implicated in cardiomyocyte hypertrophy. The present study tested the hypothesis that sympathetic-mediated hypertrophy of neonatal rat ventricular cardiomyocytes (NRVMs) tran...

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Detalles Bibliográficos
Autores principales: Al Katat, Aya, Zhao, Juan, Calderone, Angelino, Parent, Lucie
Formato: Online Artículo Texto
Lenguaje:English
Publicado: MDPI 2022
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8774121/
https://www.ncbi.nlm.nih.gov/pubmed/35053304
http://dx.doi.org/10.3390/cells11020188

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8774121/
https://www.ncbi.nlm.nih.gov/pubmed/35053304
http://dx.doi.org/10.3390/cells11020188

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