EBV(+) tumors exploit tumor cell-intrinsic and -extrinsic mechanisms to produce regulatory T cell-recruiting chemokines CCL17 and CCL22

The Epstein-Barr Virus (EBV) is involved in the etiology of multiple hematologic and epithelial human cancers. EBV(+) tumors employ multiple immune escape mechanisms, including the recruitment of immunosuppressive regulatory T cells (T(reg)). Here, we show some EBV(+) tumor cells express high levels...

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Detalles Bibliográficos
Autores principales: Jorapur, Aparna, Marshall, Lisa A., Jacobson, Scott, Xu, Mengshu, Marubayashi, Sachie, Zibinsky, Mikhail, Hu, Dennis X., Robles, Omar, Jackson, Jeffrey J., Baloche, Valentin, Busson, Pierre, Wustrow, David, Brockstedt, Dirk G., Talay, Oezcan, Kassner, Paul D., Cutler, Gene
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Public Library of Science 2022
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8791514/
https://www.ncbi.nlm.nih.gov/pubmed/35025968
http://dx.doi.org/10.1371/journal.ppat.1010200

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8791514/
https://www.ncbi.nlm.nih.gov/pubmed/35025968
http://dx.doi.org/10.1371/journal.ppat.1010200

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