Depleted HDAC3 attenuates hyperuricemia-induced renal interstitial fibrosis via miR-19b-3p/SF3B3 axis

Dysfunctional histone deacetylases (HDACs) elicit unrestrained fibrosis and damage to organs. With regard to the link between HDACs and fibrosis, this research is practiced to decipher the concrete mechanism of HDAC3 in hyperuricemia (HN)-induced renal interstitial fibrosis (RIF) from microRNA-19b-3...

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Detalles Bibliográficos
Autores principales: Hu, Langtao, Yang, Kai, Mai, Xing, Wei, Jiali, Ma, Chunyang
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Taylor & Francis 2022
Materias:
Research Paper
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8942505/
https://www.ncbi.nlm.nih.gov/pubmed/35025700
http://dx.doi.org/10.1080/15384101.2021.1989899

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8942505/
https://www.ncbi.nlm.nih.gov/pubmed/35025700
http://dx.doi.org/10.1080/15384101.2021.1989899

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