GPR65 promotes intestinal mucosal Th1 and Th17 cell differentiation and gut inflammation through downregulating NUAK2

G protein‐coupled receptor 65 (GPR65), a susceptibility gene for inflammatory bowel diseases (IBD), has been identified to promote Th17 cell pathogenicity and induce T cell apoptosis. However, the potential role of GPR65 in modulating CD4(+) T cell immune responses in the pathogenesis of IBD stills...

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Detalles Bibliográficos
Autores principales: Lin, Ritian, Wu, Wei, Chen, Huimin, Gao, Han, Wu, Xiaohan, Li, Gengfeng, He, Qiong, Lu, Huiying, Sun, Mingming, Liu, Zhanju
Formato: Online Artículo Texto
Lenguaje:English
Publicado: John Wiley and Sons Inc. 2022
Materias:
Research Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8958354/
https://www.ncbi.nlm.nih.gov/pubmed/35343079
http://dx.doi.org/10.1002/ctm2.771

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8958354/
https://www.ncbi.nlm.nih.gov/pubmed/35343079
http://dx.doi.org/10.1002/ctm2.771

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