Protein tyrosine kinase Abl promotes hepatitis C virus particle assembly via interaction with viral substrate activator NS5A

Previously, we reported that knockdown of Abl protein tyrosine kinase by shRNA or pharmacological inhibition suppresses particle assembly of J6/JFH1 strain–derived hepatitis C virus (HCV) in Huh-7.5 cells. However, the detailed mechanism by which Abl regulates HCV replication remained unclear. In th...

Descripción completa

Detalles Bibliográficos
Autores principales: Miyamoto, Daisuke, Takeuchi, Kenji, Chihara, Kazuyasu, Fujieda, Shigeharu, Sada, Kiyonao
Formato: Online Artículo Texto
Lenguaje:English
Publicado: American Society for Biochemistry and Molecular Biology 2022
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8980994/
https://www.ncbi.nlm.nih.gov/pubmed/35257746
http://dx.doi.org/10.1016/j.jbc.2022.101804

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8980994/
https://www.ncbi.nlm.nih.gov/pubmed/35257746
http://dx.doi.org/10.1016/j.jbc.2022.101804

Ejemplares similares