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Protein tyrosine kinase Abl promotes hepatitis C virus particle assembly via interaction with viral substrate activator NS5A

Previously, we reported that knockdown of Abl protein tyrosine kinase by shRNA or pharmacological inhibition suppresses particle assembly of J6/JFH1 strain–derived hepatitis C virus (HCV) in Huh-7.5 cells. However, the detailed mechanism by which Abl regulates HCV replication remained unclear. In th...

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Detalles Bibliográficos
Autores principales:	Miyamoto, Daisuke, Takeuchi, Kenji, Chihara, Kazuyasu, Fujieda, Shigeharu, Sada, Kiyonao
Formato:	Online Artículo Texto
Lenguaje:	English
Publicado:	American Society for Biochemistry and Molecular Biology 2022
Materias:	Research Article
Acceso en línea:	https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8980994/ https://www.ncbi.nlm.nih.gov/pubmed/35257746 http://dx.doi.org/10.1016/j.jbc.2022.101804

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