A glibenclamide-sensitive TRPM4-mediated component of CA1 excitatory postsynaptic potentials appears in experimental autoimmune encephalomyelitis

The transient receptor potential melastatin 4 (TRPM4) channel contributes to disease severity in the murine experimental autoimmune encephalomyelitis (EAE) model of multiple sclerosis and to neuronal cell death in models of excitotoxicity and traumatic brain injury. As TRPM4 is activated by intracel...

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Detalles Bibliográficos
Autores principales: Fearey, Brenna C., Binkle, Lars, Mensching, Daniel, Schulze, Christian, Lohr, Christian, Friese, Manuel A., Oertner, Thomas G., Gee, Christine E.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Nature Publishing Group UK 2022
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8994783/
https://www.ncbi.nlm.nih.gov/pubmed/35397639
http://dx.doi.org/10.1038/s41598-022-09875-6

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8994783/
https://www.ncbi.nlm.nih.gov/pubmed/35397639
http://dx.doi.org/10.1038/s41598-022-09875-6

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