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Rapid shear stress-dependent ENaC membrane insertion is mediated by the endothelial glycocalyx and the mineralocorticoid receptor

The contribution of the shear stress-sensitive epithelial Na(+) channel (ENaC) to the mechanical properties of the endothelial cell surface under (patho)physiological conditions is unclear. This issue was addressed in in vivo and in vitro models for endothelial dysfunction. Cultured human umbilical...

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Detalles Bibliográficos
Autores principales: Cosgun, Zülfü C., Sternak, Magdalena, Fels, Benedikt, Bar, Anna, Kwiatkowski, Grzegorz, Pacia, Marta Z., Herrnböck, Lisbeth, Lindemann, Martina, Stegbauer, Johannes, Höges, Sascha, Chlopicki, Stefan, Kusche-Vihrog, Kristina
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Springer International Publishing 2022
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8995297/
https://www.ncbi.nlm.nih.gov/pubmed/35397686
http://dx.doi.org/10.1007/s00018-022-04260-y