Parvalbumin neuroplasticity compensates for somatostatin impairment, maintaining cognitive function in Alzheimer’s disease

BACKGROUND: Patient-to-patient variability in the degree to which β-amyloid, tau and neurodegeneration impact cognitive decline in Alzheimer’s disease (AD) complicates disease modeling and treatment. However, the underlying mechanisms leading to cognitive resilience are not resolved. We hypothesize...

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Detalles Bibliográficos
Autores principales: Morrone, Christopher Daniel, Lai, Aaron Yenhsin, Bishay, Jossana, Hill, Mary Elizabeth, McLaurin, JoAnne
Formato: Online Artículo Texto
Lenguaje:English
Publicado: BioMed Central 2022
Materias:
Research
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9063209/
https://www.ncbi.nlm.nih.gov/pubmed/35501886
http://dx.doi.org/10.1186/s40035-022-00300-6

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9063209/
https://www.ncbi.nlm.nih.gov/pubmed/35501886
http://dx.doi.org/10.1186/s40035-022-00300-6

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