Ethyl pyruvate attenuates delayed experimental cerebral vasospasm following subarachnoid haemorrhage in rats: possible role of JNK pathway

The pathophysiology of delayed cerebral vasospasm (CVS) after subarachnoid haemorrhage (SAH) is multifaceted and involves endothelial apoptosis and inflammation. Ethyl pyruvate (EP) could attenuate early brain injury following SAH via anti-inflammation and inhibition of the c-Jun N-terminal kinase (...

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Detalles Bibliográficos
Autores principales: Jin, Ke, Wu, Hui, Lv, Tao, Dai, Jiong, Zhang, Xiaohua, Jin, Yichao
Formato: Online Artículo Texto
Lenguaje:English
Publicado: The Royal Society of Chemistry 2018
Materias:
Chemistry
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9078488/
https://www.ncbi.nlm.nih.gov/pubmed/35539121
http://dx.doi.org/10.1039/c7ra10801j

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9078488/
https://www.ncbi.nlm.nih.gov/pubmed/35539121
http://dx.doi.org/10.1039/c7ra10801j

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