Paternal eNOS deficiency in mice affects glucose homeostasis and liver glycogen in male offspring without inheritance of eNOS deficiency itself

AIMS/HYPOTHESIS: It was shown that maternal endothelial nitric oxide synthase (eNOS) deficiency causes fatty liver disease and numerically lower fasting glucose in female wild-type offspring, suggesting that parental genetic variants may influence the offspring’s phenotype via epigenetic modificatio...

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Detalles Bibliográficos
Autores principales: Hocher, Berthold, Lu, Yong-Ping, Reichetzeder, Christoph, Zhang, Xiaoli, Tsuprykov, Oleg, Rahnenführer, Jan, Xie, Li, Li, Jian, Hu, Liang, Krämer, Bernhard K., Hasan, Ahmed A.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Springer Berlin Heidelberg 2022
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9174141/
https://www.ncbi.nlm.nih.gov/pubmed/35488925
http://dx.doi.org/10.1007/s00125-022-05700-x

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9174141/
https://www.ncbi.nlm.nih.gov/pubmed/35488925
http://dx.doi.org/10.1007/s00125-022-05700-x

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