Cigarette smoke extract-mediated FABP4 upregulation suppresses viability and induces apoptosis, inflammation and oxidative stress of bronchial epithelial cells by activating p38 MAPK/MK2 signaling pathway

BACKGROUND: Long-term inhalation of cigarette smoke is considered to be one of the main causes of bronchial epithelioid cell damage, but its underlying mechanism has to be further clarified. METHODS: Gene expression at mRNA level and protein levels were detected by qRT-PCR and western blot analysis...

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Detalles Bibliográficos
Autores principales: Zhang, Wei, Zhang, Yibin, Zhu, Qi
Formato: Online Artículo Texto
Lenguaje:English
Publicado: BioMed Central 2022
Materias:
Research
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9202166/
https://www.ncbi.nlm.nih.gov/pubmed/35706027
http://dx.doi.org/10.1186/s12950-022-00304-z

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9202166/
https://www.ncbi.nlm.nih.gov/pubmed/35706027
http://dx.doi.org/10.1186/s12950-022-00304-z

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