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Cigarette smoke extract-mediated FABP4 upregulation suppresses viability and induces apoptosis, inflammation and oxidative stress of bronchial epithelial cells by activating p38 MAPK/MK2 signaling pathway
BACKGROUND: Long-term inhalation of cigarette smoke is considered to be one of the main causes of bronchial epithelioid cell damage, but its underlying mechanism has to be further clarified. METHODS: Gene expression at mRNA level and protein levels were detected by qRT-PCR and western blot analysis...
| Autores principales: | , , |
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| Formato: | Online Artículo Texto |
| Lenguaje: | English |
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BioMed Central
2022
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| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9202166/ https://www.ncbi.nlm.nih.gov/pubmed/35706027 http://dx.doi.org/10.1186/s12950-022-00304-z |