sGRP78 enhances selective autophagy of monomeric TLR4 to regulate myeloid cell death

Soluble glucose regulated protein 78 (sGRP78) has long been suggested as a mediator resolution of inflammation. We previously reported that sGRP78 induced the rapid endocytosis of TLR4 with defective TLR4 signaling. To elucidate the underlying mechanisms, in this study, we investigated how sGRP78 in...

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Detalles Bibliográficos
Autores principales: Wu, Zhenghao, Xu, Zhuoshuo, Zhou, Xiaoqi, Li, Heli, Zhao, Liang, Lv, Yibing, Guo, Yanyan, Shen, Guanxin, He, Yong, Lei, Ping
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Nature Publishing Group UK 2022
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9262968/
https://www.ncbi.nlm.nih.gov/pubmed/35798718
http://dx.doi.org/10.1038/s41419-022-05048-5

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9262968/
https://www.ncbi.nlm.nih.gov/pubmed/35798718
http://dx.doi.org/10.1038/s41419-022-05048-5

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