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Inhibitory immune checkpoints cause exhaustion of viral immunity in coronary artery disease
In patients with coronary artery disease, stabilizing post-translational modifications to the mRNA of the immune-checkpoint inhibitor CD155 result in an immunosuppressive macrophage phenotype and impair activation of T cells in response to viral infection.
| Formato: | Online Artículo Texto |
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| Lenguaje: | English |
| Publicado: |
Nature Publishing Group UK
2022
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| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9281274/ https://www.ncbi.nlm.nih.gov/pubmed/35856084 http://dx.doi.org/10.1038/s44161-022-00103-y |