MiR‐142‐3p regulates synaptopathy‐driven disease progression in multiple sclerosis

AIM: We recently proposed miR‐142‐3p as a molecular player in inflammatory synaptopathy, a new pathogenic hallmark of multiple sclerosis (MS) and of its mouse model experimental autoimmune encephalomyelitis (EAE), that leads to neuronal loss independently of demyelination. MiR‐142‐3p seems to be uni...

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Detalles Bibliográficos
Autores principales: De Vito, Francesca, Musella, Alessandra, Fresegna, Diego, Rizzo, Francesca Romana, Gentile, Antonietta, Stampanoni Bassi, Mario, Gilio, Luana, Buttari, Fabio, Procaccini, Claudio, Colamatteo, Alessandra, Bullitta, Silvia, Guadalupi, Livia, Caioli, Silvia, Vanni, Valentina, Balletta, Sara, Sanna, Krizia, Bruno, Antonio, Dolcetti, Ettore, Furlan, Roberto, Finardi, Annamaria, Licursi, Valerio, Drulovic, Jelena, Pekmezovic, Tatjana, Fusco, Clorinda, Bruzzaniti, Sara, Hornstein, Eran, Uccelli, Antonio, Salvetti, Marco, Matarese, Giuseppe, Centonze, Diego, Mandolesi, Georgia
Formato: Online Artículo Texto
Lenguaje:English
Publicado: John Wiley and Sons Inc. 2021
Materias:
Original Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9291627/
https://www.ncbi.nlm.nih.gov/pubmed/34490928
http://dx.doi.org/10.1111/nan.12765

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9291627/
https://www.ncbi.nlm.nih.gov/pubmed/34490928
http://dx.doi.org/10.1111/nan.12765

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