Ischemic damage to every segment of the oxidative phosphorylation cascade elevates ETC driving force and ROS production in cardiac mitochondria

Myocardial ischemia has long-lasting negative impacts on cardiomyocyte mitochondrial ATP production. However, the location(s) of damage to the oxidative phosphorylation pathway responsible for altered mitochondrial function is unclear. Mitochondrial reactive oxygen species (ROS) production increases...

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Detalles Bibliográficos
Autores principales: Kuzmiak-Glancy, Sarah, Glancy, Brian, Kay, Matthew W.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: American Physiological Society 2022
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9448280/
https://www.ncbi.nlm.nih.gov/pubmed/35867709
http://dx.doi.org/10.1152/ajpheart.00129.2022

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9448280/
https://www.ncbi.nlm.nih.gov/pubmed/35867709
http://dx.doi.org/10.1152/ajpheart.00129.2022

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