Rhein ameliorates transverse aortic constriction-induced cardiac hypertrophy via regulating STAT3 and p38 MAPK signaling pathways

The progression from compensatory hypertrophy to heart failure is difficult to reverse, in part due to extracellular matrix fibrosis and continuous activation of abnormal signaling pathways. Although the anthraquinone rhein has been examined for its many biological properties, it is not clear whethe...

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Detalles Bibliográficos
Autores principales: Li, Run-Jing, Xu, Jia-Jia, Zhang, Zheng-Hao, Chen, Min-Wei, Liu, Shi-Xiao, Yang, Cui, Li, Yan-Ling, Luo, Ping, Liu, Yi-Jiang, Tang, Rong, Shan, Zhong-Gui
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Frontiers Media S.A. 2022
Materias:
Pharmacology
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9459036/
https://www.ncbi.nlm.nih.gov/pubmed/36091816
http://dx.doi.org/10.3389/fphar.2022.940574

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9459036/
https://www.ncbi.nlm.nih.gov/pubmed/36091816
http://dx.doi.org/10.3389/fphar.2022.940574

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