Interleukin-17A influences the vulnerability rather than the size of established atherosclerotic plaques in apolipoprotein E-deficient mice

Interleukin (IL)-17A plays a role in the development of atherosclerotic plaques; however, the mechanism remains unclear. In this study, apolipoprotein E-deficient (ApoE(–/–)) mice were fed a high-fat diet to induce atherosclerosis, followed by the treatment with exogenous recombinant IL-17A or the n...

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Detalles Bibliográficos
Autores principales: Wang, Bo, Hou, Xitan, Sun, Yaning, Lei, Chao, Yang, Sha, Zhu, Yao, Jiang, Yingming, Song, Li
Formato: Online Artículo Texto
Lenguaje:English
Publicado: De Gruyter 2022
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9462543/
https://www.ncbi.nlm.nih.gov/pubmed/36133421
http://dx.doi.org/10.1515/biol-2022-0072

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9462543/
https://www.ncbi.nlm.nih.gov/pubmed/36133421
http://dx.doi.org/10.1515/biol-2022-0072

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