Activating transcription factor 3 inhibits angiotensin II-induced cardiomyocyte viability and fibrosis by activating the transcription of cysteine-rich angiogenic protein 61

Depletion of activating transcription factor 3 (ATF3) expression has previously been reported to promote hypertrophy, dysfunction and fibrosis in stress overload-induced hearts; however, the mechanism involved remains poorly understood. In the present study, the mechanism underlying the activation o...

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Detalles Bibliográficos
Autores principales: Zhang, Yu, Wu, Heming, Luo, Honghui, Luo, Yiqun, Huang, Cong
Formato: Online Artículo Texto
Lenguaje:English
Publicado: D.A. Spandidos 2022
Materias:
Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9494611/
https://www.ncbi.nlm.nih.gov/pubmed/36102309
http://dx.doi.org/10.3892/mmr.2022.12852

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9494611/
https://www.ncbi.nlm.nih.gov/pubmed/36102309
http://dx.doi.org/10.3892/mmr.2022.12852

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