Ac2‑26 alleviates hepatic ischemia‑reperfusion injury based on inhibiting the positive feedback loop of HMGB1/TLR4/NF‑κB/neutrophils

Inflammation is one of the most crucial mechanism underlying hepatic ischemia-reperfusion injury (HIRI). Several studies have shown that Ac2-26, the active N-terminal peptide of Annexin A1, could modulate anti-inflammatory processes and protect the organs from ischemia-reperfusion injury (IRI). Howe...

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Detalles Bibliográficos
Autores principales: Bai, Chen, Jiang, Zhengchen, Jiang, Hongxin, Yu, Shuna, Li, Ming, Chu, Fangfang, Sheng, Yaxin, Li, Jianguo, Jiang, Jiying, Li, Wanzhen
Formato: Online Artículo Texto
Lenguaje:English
Publicado: D.A. Spandidos 2022
Materias:
Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9500483/
https://www.ncbi.nlm.nih.gov/pubmed/36237600
http://dx.doi.org/10.3892/etm.2022.11609

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9500483/
https://www.ncbi.nlm.nih.gov/pubmed/36237600
http://dx.doi.org/10.3892/etm.2022.11609

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